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Staffing & Compensation Plan

Please ensure this CLC – Staffing & Compensation Plan Sample Essay Papers work has scholarly references; free of plagiarism, free of grammatical error. Ensure all free of incorrect spelling.

CLC – Staffing & Compensation Plan Sample Essay Papers is a Collaborative Learning Community (CLC) assignment.

Develop a 750-1,000 word high level staffing and compensation plan. The plan should contain an introduction and conclusion that make relevant connections to the course objectives.

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The purpose of this CLC – Staffing & Compensation Plan Sample Essay Papers assignment is to simulate the thought process that managers use when planning for staffing and compensating employees Staffing & Compensation Plan. Students are not expected to produce a detailed proposal. Rather, the expectation is that students demonstrate consideration of the multiple complexities involved in staffing and compensation.

Obtain the Instructor’s approval on the selected organization before writing the Staffing and Compensation Plan which should include:

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Introduction.

Discussion of the staffing needs of your organization.

Explanation of the compensation plan for your organization.

Description of the employee benefits offered.

Conclusion.

Incorporate three to five resources (including your textbook) to support your Staffing and Compensation Plan.

Prepare this assignment according to the guidelines found in the APA Style Guide, located in the Student Success Center. An abstract is not required.

This assignment uses a rubric. Please review the rubric prior to beginning the assignment to become familiar with the expectations for successful completion.

You are required to submit this
CLC – Staffing & Compensation Plan
Sample Essay Papers
assignment
to Turnitin. Please refer to the directions in the Student Success Center. Staffing & Compensation Plan.

Power use and misuse essay example
.

TS Eliot wrote, “Half of the harm that is done in this world is caused by people who have power and want to feel important. They do not mean to do harm… they are simply absorbed in the useless struggle to think well of themselves.” (Manning & Curtis , p. 167)

Please do Exercise 8-1, p. 169. What kind of power does your supervisor commonly use, and what has been the outcome of their behavior…on the employees, on the company?

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Exercise 8–1 What Type of Power Does Your Supervisor Use?

Indicate how strongly you agree or disagree with the following statements as they describe your immediate supervisor. If you are not currently employed, evaluate a supervisor you have had in the past. For each statement, select the most appropriate response, using the following scale: 1 = strongly disagree; 2 = disagree; 3 = neither agree nor disagree; 4 = agree; 5 = strongly agree.

My supervisor 1
.

1
.   ______ Recognizes efforts and rewards accomplishments.

2
.   ______ Uses fear to control behavior.

3
.   ______ Shows appreciation for work well done.

4
.   ______ Manipulates others to gain compliance.

5
.   ______ Makes timely decisions to get things done.

6
.   ______ Stays abreast of job-related news.

7
.   ______ Exercises authority and power of position.

8
.   ______ Provides skill and advice to solve job problems.

9
.   ______ Maintains access to important facts and data.

10
.   ______ Keeps job knowledge current.

11
.   ______ Has high ideals and standards of conduct.

12
.   ______ Thinks clearly and explains things logically.

13
.   ______ Causes people to respect what he or she stands for.

14
.   ______ Creates a vision and strong sense of purpose.

15
.   ______ Gets the facts before making decisions.

16
.   ______ Motivates others and inspires them to action. Power use and misuse essay example

Scoring:

Power of the Position

1
.   Add the numbers assigned to statements 1 and 3. This is the 
reward power
________

2
.   Add the numbers assigned to statements 2 and 4. This is the 
coercive power
________

3
.   Add the numbers assigned to statements 5 and 7. This is the 
legitimate power
________

4
.   Add the numbers assigned to statements 6 and 9. This is the 
information power
________

Power of the Person

5
.   Add the numbers assigned to statements 8 and 10. This is the 
expert power
________

6
.   Add the numbers assigned to statements 11 and 13. This is the 
referent power
________

7
.   Add the numbers assigned to statements 12 and 15. This is the 
rational power
________

8
.   Add the numbers assigned to statements 14 and 16. This is the 
charisma power
________

Discussion:

The effective leader emphasizes the power of the person to accomplish goals. This involves maintaining knowledge and skill (expertise), having high moral character (referent power), demonstrating effective problem-solving ability (rational power), and motivating and inspiring people (charisma). The effective leader also uses the power of the position to reward efforts and accomplishments (rewards), make effective decisions (legitimacy), and keep people informed on important matters (information). The effective leader rarely if ever uses fear (coercion) as a form of power. Coercion involves threats, punishment, and negative rewards. It usually leads to negative feelings of resentment and counterproductive work behavior—complaints, rebellion, and reduced productivity.

Power

Exercise 8–1 What Type of Power Does Your Supervisor Use?

Indicate how strongly you agree or disagree with the following statements as they describe your immediate supervisor Power use and misuse essay example. If you are not currently employed, evaluate a supervisor you have had in the past. For each statement, select the most appropriate response, using the following scale: 1 = strongly disagree; 2 = disagree; 3 = neither agree nor disagree; 4 = agree; 5 = strongly agree.

My supervisor 1
.

1
.   
4
Recognizes efforts and rewards accomplishments.

2
.   
2
Uses fear to control behavior.

3
.   
4
Shows appreciation for work well done.

4
.   
2
Manipulates others to gain compliance.

5
.   
3
Makes timely decisions to get things done.

6
.   
3
Stays abreast of job-related news.

7
.   
5
Exercises authority and power of position.

8
.   
4
Provides skill and advice to solve job problems.

9
.   
4
Maintains access to important facts and data.

10
.   
4
Keeps job knowledge current.

11
.   
4
Has high ideals and standards of conduct.

12
.   
4
Thinks clearly and explains things logically.

13
.   
4
Causes people to respect what he or she stands for.

14
.   
4
Creates a vision and strong sense of purpose.

15
.   
4
Gets the facts before making decisions.

16
.   
4
Motivates others and inspires them to action.

Scoring:

Power of the Position

1
.   Add the numbers assigned to statements 1 and 3. This is the 
reward power

8

2
.   Add the numbers assigned to statements 2 and 4. This is the 
coercive power

4

3
.   Add the numbers assigned to statements 5 and 7. This is the 
legitimate power

8

4
.   Add the numbers assigned to statements 6 and 9. This is the 
information power

7

Power use and misuse essay example

Power of the Person

5
.   Add the numbers assigned to statements 8 and 10. This is the 
expert power

8

6
.   Add the numbers assigned to statements 11 and 13. This is the 
referent power

8

7
.   Add the numbers assigned to statements 12 and 15. This is the 
rational power

8

8
.   Add the numbers assigned to statements 14 and 16. This is the 
charisma power

8

What kind of power does your supervisor commonly use, and what has been the outcome of their behavior…on the employees, on the company?

My supervisor appears to use more of power of the person rather than power of the position. That is because she scores more highly in the power of person scores when compared to the power of position scores Power use and misuse essay example. In fact, she is an effective leader who has much expertise to include knowledge and skills having scored 8 out of 10. Additionally, she has a high moral character with a score of 8 out of 10. Also, she makes good use of rational power with a good problem-solving capacity having scored 8 out of 10. Besides that, she is charismatic with the capacity for motivation and inspiration with a score of 8 out of 10.

With regards to use of the power of position, my supervisor appears be an effective leader. She scores 8 out of 10 in reward power, by rewarding her subordinates efforts and accomplishments. Also, she has legitimacy as a leader by scoring 8 out of 10 in making effective decisions. Additionally, she makes very little use of coercive power, scoring 4 out of 10 to show that she rarely uses fear, negative rewards, punishment and threats against her subordinates. Furthermore, she has a score of 7 out of 10 with regards to information power, showing that she jeeps people informed about important matters. Power use and misuse essay example. In essence, she is an effective leader as evidenced when she makes extensive use of power of the person although she could improve her performance in this regard by increasing the scores from 8 to 10. She could also improve her power of position by increasing her reward, legitimate and information power scores (getting them to 10) even as she reduces her coercive power score (getting it to 1).

As a person in a position of power, my supervisor has invested in the development of her leadership skills. As her subordinate, I feel that I am valued and directed to work more effectively in ensuring that the organization goals are met. Also, I feel that I am attracted to remain her subordinate and feel the desire to stay loyal to her. That is because she recognizes my input, focuses on the well-being and growth of me as a subordinate and the work environment even as she engenders as sense of belonging. Her functional attributes are individualized consideration, intellectual stimulation, inspirational motivation, charisma, empowerment, appreciation of others, modeling, service, trust, integrity, honesty, and vision. This is contrasted to traditional leadership where the leader’s primary focus is on power of the position and less on power of the person in exercising the accumulated power to be at the top (Manning & Curtis, 2003; Marquis & Huston, 2014). In this respect, my supervisor is an effective leader Power use and misuse essay example.

 

References

Manning, G. & Curtis, K. (2003).
The Art of Leadership
. New York, NY: McGraw Hill Professional.

Marquis, B. & Huston, C. (2014).
Leadership Roles and Management Functions in Nursing: Theory and application
(8
th
ed.). Philadelphia, PA: Lippincott, Williams & Wilkins Power use and misuse essay example.

Endocrine System Essay

Pre-Lab Evaluation Answers.

The endocrine system produces hormones to regulate the internal environment of the body therefore keeping the parameters of the body at a relatively constant level. The hormones work by a feedback mechanism whereby if a parameter goes high or low it stimulates the production of the hormone that is responsible for regulating it back to the normal level. This is important so that the cells are at the environment at which they work best Endocrine System Essay.

Parathyroid hormone, a hormone responsible for calcium regulation, is produced by the parathyroid glands. They are two glands and are located on the posterior aspect of the thyroid glands. Secretion of parathyroid hormone is inversely related to serum calcium level. The direct action of parathyroid hormone is on bone and kidneys. It causes release of calcium from the bone and reabsorption in the kidneys thereby raising the level of calcium in blood (Lok, U, 2014).

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On the anterior aspect, the chief cells of the thyroid glands produce calcitonin. Calcitonin is antagonistic to parathyroid hormone and functions to reduce the amount of calcium in blood.

 

The two hormones secreted by the thyroid gland are T3 and T4, they are important in regulating the metabolism of the body. A high level of T3 and T4 results in high metabolism. The difference between them is that T3 has three molecules while T4 has four. T3 is the more active one. They require iodine in their structure (Mullur, R, 2014).

Corticosteroids are a group of drugs/hormones that resemble in structure the hormones that are produced by the cortex of the adrenal gland. An example is prednisone. They are mostly used to treat autoimmune inflammatory diseases. Their effects include hyperglycemia and weight gain; they also depress the hypothalamo-pituitary-adrenal axis Endocrine System Essay.

Adequate blood sugar levels are essential so that cells can be able to take in the sugar to perform their metabolic functions. Insulin is the hormone that is responsible for transporting glucose into the cells. Normal blood sugar levels in humans are 3.9-5.4 mmols/l for fasting blood sugar and 5.5-7.0 mmols/l for random blood sugar. The pancreas produces insulin to reduce the blood sugar when it is high, while the liver stores glycogen, which is converted into glucose to raise the blood when it is low (Hill,N, 2011).

Post-Lab Evaluation Answers

The significance of the HbA1c test is to determine whether blood sugar control has been adequate over the past 3 months. In Mr Joe’s case it has been poor. The diagnosis is poorly controlled diabetes mellitus Endocrine System Essay.

If Joe were honest he most likely responded by saying that he has not been watching his diet. I would explain to Joe that Diabetes is a chronic disease that results from either inadequate or lack of insulin and that it is not cured by medication but is instead controlled by a lifetime of oral or injectable drugs that are essential to control his blood sugar.

I would explain to Joe that there are different methods of controlling his blood sugar. I would start by advising him to pursue a change in diet and lose some weight and also advice him that he does not have to be on insulin shots if his blood sugar control is adequately controlled by oral medication.

In the short term Joe could go into diabetic ketoacidosis, a hyperosmolar state or hypoglycemic coma. In the long term Joe could have vision loss, heart disease, kidney disease, peripheral neuropathy and leg ulcers.

I would advice Joe to talk to his son about diet modification and weight loss. And also for him to do follow the same advice. Endocrine System Essay.

References

Lok, U., Hatipoglu, S., Gulacti, U., Arpaci, A., Aktas, N., & Borta, T. (2014). The Role of Thyroid and Parathyroid Metabolism Disorders in the Etiology of Sudden Onset Dizziness.
Medical Science Monitor : International Medical Journal of Experimental and Clinical Research
,
20
, 2689–2694. http://doi.org/10.12659/MSM.891305

Mullur, R., Liu, Y.-Y., & Brent, G. A. (2014). Thyroid Hormone Regulation of Metabolism.
Physiological Reviews
,
94
(2), 355–382.
http://doi.org/10.1152/physrev.00030.2013

Hill, N. R., Oliver, N. S., Choudhary, P., Levy, J. C., Hindmarsh, P., & Matthews, D. R. (2011). Normal Reference Range for Mean Tissue Glucose and Glycemic Variability Derived from Continuous Glucose Monitoring for Subjects Without Diabetes in Different Ethnic Groups.
Diabetes Technology & Therapeutics
,
13
(9), 921–928. http://doi.org/10.1089/dia.2010.0247 Endocrine System Essay

Financial healthcare management essay – CPT code 99214

Case Study: You are assessing the payer–patient mix for a health care organization. Currently, your payer mix is 40% Medicare, 10% Medicaid, 25% traditional indemnity insurance, 20% managed care, and 5% self-pay patients. Complete the following:

Using the most common office visit, CPT code 99214, determine the reimbursement from the Centers for Medicare and Medicaid Services (online fee schedule available for Medicare).

Using the same CPT code, 99214, determine the be possible to obtain actual reimbursement information from your personal insurance carrier. If the information is not available, assume reimbursement by traditional indemnity insurance is usually 200% reimbursed more than Medicare and Medicaid, and managed care is usually 133% more than Medicare and Medicaid.

Compose an accounts receivable benchmark from this information showing columns for current, 30–60, 61–90, 90–120, and greater than 120 days. Financial healthcare management essay – CPT code 99214.

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Assess the information for areas of improved reimbursement of at least 20% or more.

Evaluate the options available to change the payer–patient mix with consideration of related legal and ethical issues.

Propose a best strategy with justification and rationale based on effective decision-making tenets.

Spreadsheets demonstrating the accounts receivable analysis will be attached as appendices, and there will be at least 7 relevant peer-reviewed academic or professional references published within the past 5 years. Also attach appropriate fee schedules. Financial healthcare management essay – CPT code 99214.

Developing advocacy campaign – Child Obesity Essay

To prepare:

Select a population health issue of interest to you and identify the population affected by the issue.

Locate two scholarly articles, each of which provides a description of an effective health advocacy campaign that addresses your issue. The articles need to focus on two different advocacy campaigns.

Analyze the attributes of the two campaigns to determine what made them effective.

Reflect on a policy you could propose or suggest a change to a current policy to improve the health of the population you selected.

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Consider how you could develop an advocacy campaign, applying the attributes identified in similar, effective campaigns.

To complete:

For the Part 1 application (approximately 3 pages of content with a title page and references in APA format) address the following:

Describe your selected population health issue and the population affected by this issue.

Summarize the two advocacy campaigns you researched in this area.

Explain the attributes that made those campaigns effective.

Begin to develop a plan for a health advocacy campaign that seeks to create a new policy or change an existing policy with regard to the issue and population you selected. Be sure to include in your plan:

A description of the public health issue and proposed policy solution Developing advocacy campaign – Child Obesity Essay.

Specific objectives for the policy you want to be implemented

Begin to substantiate of your proposed campaign by data and evidence.

 

Developing an Advocacy Campaign

Introduction

The CDC (2015) reports that close to 13 million children in the USA (17% of their population) are suffering from obesity, with that population including individuals between 2 and 19 years of age. It is caused by changes in physical activity and dietary patterns that limit physical activity even as fatty foods consumption is increased Developing advocacy campaign – Child Obesity Essay. This is based on the understanding that obesity is by a body mass index (BMI) figure that exceeds 30 in which fat accumulates in the body to present both social implications, health risks, and economic effects. The social implications include: deleterious self-image; diminished self-esteem; suicidal tendencies; low self-esteem; low body satisfaction; eating disorders; and reduced quality of life. The health risks include expression of: type 2 diabetes; compromised glucose tolerance; apnea; stress; gout; gallstones; fatty liver; musculoskeletal disorders like osteoarthritis; hypertension; dyslipidemia; predisposition to cardiovascular diseases; pregancy complications when older; and cancers of the endometrium, breast and colon (Childhood Obesity Foundation, 2015). The economic effects take the form of direct costs, intangible costs and indirect costs to include; medical services; hospital-related costs; medication; and productivity losses (Katz et al., 2014). Therefore, childhood obesity is a source of medical and social concern that must be addressed as a priority within the USA.

Objective

Medical systems are intended to ensure that populations are healthy. In this case, childhood obesity has been identified as a source of concern that must be addressed with the intention of reducing its incidence. As such, the main objective of this paper is to identify the for an intervention plan regarding childhood obesity, as well as develop a feasible advocacy plan for childhood obesity Developing advocacy campaign – Child Obesity Essay.

Proposed Campaign by Evidence

There is a consensus that lack of government support has created a situation in which the society and environment creates conditions that are unfavorable for good diet and physical activity among children thereby allowing them to develop obesity. In essence, the lack of supportive government policies has created ideal conditions for the continued presentation of childhood obesity as a source of concern within the public arena. The federal government is aware of this concern and has since taken steps to address the issue. Key in the government efforts is the Healthy, Hunger-Free Kids Act of 2010 that provides funding for school-focused national programs targeted at addressing the occurrence of obesity within the school children. Working through the Act, the government has been able to conceptualize, present, fund and support school feeding programs whereby school diets are optimized to ensure that schoolchildren are fed with healthy food. In fact, the program has adopted a school-focused approach, making it possible for school to provide their students with foods that have the correct nutrient values. One of the campaigns that the Act has supported is the Coordinated School Health Program (CSHP). The program focuses on eight interrelated and critical modules. The eight modules include involvement of the community and family, environmental safety and health, nutrition services, social services, health services, physical education and health education (King & Ling, 2015).

Other than the school-focused programs, there are advocacy campaigns that have applied a family-focused approach. One of this is the Let’s Move campaign that has identified the family unit as a valuable tool for addressing childhood obesity thereby adopting a family-focused approach. The campaign is based on the understanding that the readiness and motivation to change behavior must start in the family unit (Bumpus, Tagtow & Haven, 2015). Childhood Obesity Foundation (2015) identifies family-focused approach, as applied by Let’s Move, as a tool for bringing about effective and active lifestyle changes for the family as a unit and children as part of the unit that is influenced by the setting.

The two campaigns make it clear that any attempt targeted at addressing childhood obesity as a health concern must actively involve the community of which the children form a part to include the schools and family. They are grounded on the idea that healthy behavior is resultant of dynamic links between the social environment and influences realized from interpersonal, organization, community and policy level. This means that social, group and individual efforts should be combined to bring about behavioral change within the community as required for effective management of childhood obesity (Cohen et al., 2014). This makes it evident that any campaign targeted at reducing the incidence of childhood obesity must include social, group, and individual efforts. They use social marketing as a strategy to promote health behavior change, drawing on concepts that are typically applied in business marketing and modifying them to be used in promoting socially desirable health-related behaviors that include eating the right food and being physically active. They can use health workers to counsel the community members or even addresses in town meetings (McKenzie-Mohr, 2013). A prominent advantage of social marketing use in implementing community behavior change is that it introduces health promotion behaviors that do not rely on messages concerning health risk, and severity, as is the case in other approaches applied to implement community behavioral changes (Katz et al., 2014). Based on this analysis, the proposal is for the use of an advocacy plan that combines elements from both school-focused and family-focused campaigns to address childhood obesity. Amalgamating the two programs will address different populations with the same goal, thereby improving the possibility for success if they are used in combination.

References

Bumpus, K., Tagtow, A. & Haven, J. (2015). Let’s Move! Celebrates 5 Years.
Journal of the Academy of Nutrition and Dietetics
, 115(3), 338-341.

CDC (2015).
Childhood Obesity Facts
. Retrieved from https://www.cdc.gov/obesity/data/childhood.html

Childhood Obesity Foundation (2015).
Childhood Obesity
. Retrieved from http://www.childhoodobesityfoundation.ca/whatIsChildhoodObesity

Cohen, J., Richardson, S., Parker, E., Catalano, P. & Rimm, E. (2014). Impact of the New U.S. Department of Agriculture School Meal Standards on Food Selection, Consumption, and Waste.
Am J Prev Med.
, 46(4), 388-394.

Katz, D., Elmore, J., Wild, D. & Lucan, S. (2014).
Jekel’s Epidemiology, Biostatistics, Preventive Medicine, and Public Health
(4
th
ed.). Philadelphia, PA: Elsevier Saunders.

King, K. & Ling, J. (2015). Results of a 3-year, nutrition and physical activity intervention for children in rural, low-socioeconomic status elementary schools.
Health Education Research
, 30(4), 647-659.

McKenzie-Mohr, D. (2013).
Fostering Sustainable Behavior: An introduction to community-based social marketing
(3
rd
ed.). Gabriola Island, British Columbia: New Society Publishers.

 

In the first assignment, you reflected on whether the policy you would like to promote could best be achieved through the development of new legislation, or a change in an existing law or regulation. Refine as necessary using any feedback from your first Developing advocacy campaign – Child Obesity Essay paper.

Contemplate how existing laws or regulations may affect how you proceed in advocating for your proposed policy.

Consider how you could influence legislators or other policymakers to enact the policy you propose.

Think about the obstacles of the legislative process that may prevent your proposed policy from being implemented as intended.

To complete:

Part Two will have approximately 3  pages of content plus a title page and references. Part Two will address the following:

Explain whether your proposed policy could be enacted through a modification of existing law or regulation or the creation of new legislation/regulation.

Explain how existing laws or regulations could affect your advocacy efforts. Be sure to cite and reference the laws and regulations using primary sources.

Provide an analysis of the methods you could use to influence legislators or other policymakers to support your policy. In particular, explain how you would use the “three legs” of lobbying in your advocacy efforts Developing advocacy campaign – Child Obesity Essay.

Summarize obstacles that could arise in the legislative process and how to overcome these hurdles.

Advocacy Campaign

Introduction

It is acknowledged that childhood obesity is a source of social, medical and financial concerns since it affects 17% of this population (Centers for Disease Control and Prevention, 2015). In fact, the social concerns include deleterious self-image, diminished self-esteem, suicidal tendencies, low self-esteem, low body satisfaction, eating disorders, and reduced quality of life. The health concerns include predisposition to type 2 diabetes, compromised glucose tolerance, apnea, stress, gout, gallstones, fatty liver, musculoskeletal disorders like osteoarthritis, hypertension, dyslipidemia, predisposition to cardiovascular diseases, pregnancy complications when older, and cancers of the endometrium, breast and colon (Childhood Obesity Foundation, 2015). The economic concerns include direct, intangible and indirect costs linked with medical services, hospital-related costs, medication, and productivity losses (Katz et al., 2014). Following this awareness, there is a need for an advocacy plan that facilitates the presentation of a healthy diet while encouraging physical activity to preempt and reverse childhood obesity. The proposed advocacy plan intends to achieve this goal by using elements of both family-focuses and school-focused approaches the entail implementing a two pronged approach that targets children both at school and at home where they have the most social interactions Developing advocacy campaign – Child Obesity Essay.

Explain whether your proposed policy could be enacted through a modification of existing law or regulation or the creation of new legislation/regulation.

The proposed policy will be enacted by modifying an existing legislation. In this case, the Healthy, Hunger-Free Kids Act of 2010 is intended for revision. As the Act stands, it advocates for a school-focused approach by allocated federal resources towards conceptualizing, presenting, funding and supporting school feeding programs across the USA. Through the Act’s mandate, school diets have been optimized to offer healthy foods to school children while also considering their nutrient needs. Although the Act has comprehensively improved obesity management in school environments through involving the community, environmental safety and health, nutrition services, social services, health services, physical education and health education (King & Ling, 2015), it can be faulted for only applying half measures by focusing on one social environment when there are many environments that should be addressed for a better outcomes. In fact, the campaign identifies the family as a social environment in which the children spend most of their time and take many meals. In fact, a typical school child will take two major meals at home (breakfast and dinner) on a typical school day while only taking one major meal in school (lunch). The implication is that the current framing of the Healthy, Hunger-Free Kids Act of 2010 only has a direct influence on lunch as a major meal to imply that any gains made in school could easily be overturned by detriments in the breakfast and dinner meals presented at home. Besides the meals, the family unit has an influence on the child’s readiness and motivation to change behavior and adopt a healthier lifestyle. As such, there is a need for the Act to be revised to include provisions for a family-focused approach that target the family as a tool for influencing the school children to adopt healthy lifestyles.

Explain how existing laws or regulations could affect your advocacy efforts.

Revising the Healthy, Hunger-Free Kids Act of 2010 will affect the advocacy efforts by availing resources for the plan. The Act makes provisions for federal funding to be allocated towards anti-obesity campaigns (King & Ling, 2015). Using the revised Act, the advocacy will gain access to the extensive government resources (including funding, personnel, linkages, and so on) to roll out nationwide with minimal resistance. This means that should the Act be revised to include a family-focused approach along with the school-focused approach, then its roll out would be easily implemented by the federal government.

Provide an analysis of the methods you could use to influence legislators or other policymakers to support your policy. In particular, explain how you would use the “three legs” of lobbying in your advocacy efforts.

The “three legs” of lobbying will be applied in seeking support for revising Healthy, Hunger-Free Kids Act of 2010 from legislators and policymakers. Firstly, direct lobbying will be applied by convincing professional health bodies and organizations concerned with childhood obesity to gain their support. For instance, support will be sought from the American National Association since the advocacy plan has been conceptualized by a nurse who is a member of the organization. Secondly, grassroots lobbying will be applied by rallying communities to support the revision through presenting them with verified tangible data that shows the merits of the plan. This would include holding community forums and discussions where the community members are allowed to question the plan even as it is presented. Finally, political lobbying will be applied by seeking help from legislators to place the revision through the legislative process. This would entail framing the revision in a realistic manner that does not conflict with other existing legislation then getting legislators to endorse its passage (Goodwin, K., Ainsworth & Goodwin, 2013). Applying the three approaches to lobbying is anticipated to influence opinion and gain support for the advocacy plan Developing advocacy campaign – Child Obesity Essay.

Summarize obstacles that could arise in the legislative process and how to overcome these hurdles.

A major obstacle is the laws concerning the ethics of lobbying. There are fine guidelines governing how lobbying is conducted when seeking support for a legislative change. To overcome this hurdle, the rules governing how lobbying is conducted will be reviewed with focus being on credibility (Goodwin, K., Ainsworth & Goodwin, 2013). Another obstacle is the need for funding to carry out the lobbying. Funds are necessary for coordinating support. This obstacle will be overcome by presenting a proposal to professional organizations who would then fund the lobbying efforts Developing advocacy campaign – Child Obesity Essay.

 

References

Centers for Disease Control and Prevention (2015).
Childhood Obesity Facts
. Retrieved from https://www.cdc.gov/obesity/data/childhood.html

Childhood Obesity Foundation (2015).
Childhood Obesity
. Retrieved from http://www.childhoodobesityfoundation.ca/whatIsChildhoodObesity

Goodwin, K., Ainsworth, S. & Goodwin, E. (2013).
Lobbying and Policymaking: The public pursuit of private interests
. Thousand Oaks, CA: SAGE Publications.

Katz, D., Elmore, J., Wild, D. & Lucan, S. (2014).
Jekel’s Epidemiology, Biostatistics, Preventive Medicine, and Public Health
(4
th
ed.). Philadelphia, PA: Elsevier Saunders.

King, K. & Ling, J. (2015). Results of a 3-year, nutrition and physical activity intervention for children in rural, low-socioeconomic status elementary schools.
Health Education Research
, 30(4), 647-659. Developing advocacy campaign – Child Obesity Essay.

Pathophysiology of Disorders sample assignment

Assignment 2: The Pathophysiology of Disorders

During the last 5 weeks, you have explored various body systems: neurological, cardiovascular, respiratory, and hematological. These four systems work together along with other body systems to complete a myriad of functions Pathophysiology of Disorders sample assignment. For this reason, when disorders occur within one body system, it can create potentially devastating effects throughout the entire body. For instance, Parkinson’s disease is a disorder of the central nervous system, yet its alterations actually affect multiple body systems from the cardiovascular system to the gastrointestinal system. In this Assignment, you examine alterations associated with disorders, as well as the impact of the alterations on multiple body systems.

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To Prepare

From the list below, select a disorder of interest to you:

Alzheimer’s disease

Asthma in children

Chronic obstructive pulmonary disease (COPD)

Congestive heart failure

Hepatic disease (liver disease)

Hypertension

Hyperthyroidism and hypothyroidism

Seizures

Sepsis

Identify alterations associated with your selected disorder. Consider the pathophysiology of the alterations. Think about how these alterations produce pathophysiological changes in at least two body systems.

Reflect on how patient factors such as genetics, gender, ethnicity, age, and behavior might impact the pathophysiology of the alterations you identified, as well as the diagnosis and treatment of your selected disorder.

Review the “Mind maps—Dementia, Endocarditis, and Gastro-oesophageal Reflux Disease (GERD)” media in the Week 2 Learning Resources. Use the examples in the media as a guide to construct a mind map for the disorder you selected. Consider the epidemiology and clinical presentation of your selected disorder.

To Complete

Develop a 5- to 10-slide PowerPoint presentation that addresses the following:

Describe your selected disorder, as well as associated alterations. Explain the pathophysiology of the alterations, including changes that occur in at least two body systems Pathophysiology of Disorders sample assignment.

Explain how genetics, gender, ethnicity, age, and behavior might impact the pathophysiology of the alterations you identified, as well as diagnosis and treatment of your selected disorder.

Construct a mind map for the disorder you selected. Include the epidemiology, pathophysiology of alterations, risk factors, and clinical presentation, as well as the diagnosis and treatment of the disorder.

Notes

Please follow the rubric that will be uploaded.

References must be 5 years or less

 

Pathophysiology of Disorders

Hypertension

Pathophysiology of Disorders sample assignment

Description

Hypertension is the elevation of blood pressure

Hypertension is diagnosed when the blood pressure is above 130/90 mm of mercury

Hypertension is primarily affects the heart and the vascular system

Hypertension is primarily composed of two elements;

increased cardiac output and peripheral vascular resistance (Berkman, Kawachi, & Glymour, 2014).

Blood pressure is the pressure exerted on the walls of the blood vessels by the blood flowing through them. Therefore, the amount of the blood and the counter force by the vasculature determine the pressure.

Pathophysiology

In hypertension , reduced renal perfusion stimulates renin angiotensin system.

Angiotensin II causes vasoconstriction, increasing peripheral vascular pressure

Angiotensin II also stimulates aldosterone production Pathophysiology of Disorders sample assignment

Aldosterone increases retention of water and sodium in the kidney

The blood volume is thus, increased.

Hypertension is also caused by inappropriate neuronal control of blood pressure.

The baroreceptors on the walls of blood vessels are hyperreponsive to the blood pressure.

Vascular constriction increases the peripheral vascular resistance.

dysfunction of endothelins nitric oxide causes increased blood pressure

The RAAS increases the blood volume through renal water and sodium retention mechanisms. The neuronal mechanism causes unnecessary constriction, increasing peripheral vascular pressure. The endothelins fail to cause vasodilation. Pathophysiology of Disorders sample assignment.

Body Systems

In hypertension, the endothelin system dysfunctions and;

Does not produce enough nitric oxide.

Consequently, sufficient vasodilation does not occur.

Hypertension, affects several body system.

One of the systems is the renal system.

In hypertension, the renal perfusion is impaired.

Kidneys receive less oxygenation that the demand.

In addition, the amount of blood to the kidneys is reduced.

The kidneys receive less water and blood ions.

Consequently, the amount of water and ions filtered (Wenzel, Bode, Köhl, & Ehmke, 2016).

The glomerular filtration rate is dependent on the amount of blood supply to the kidneys per unit time. Reduced blood supply results into reduced glomerular filtration rate.

Body Systems cont’d

The kidney excretion is reduced, thus, reduced glomerular filtration rate.

Hypertension affects the brain.

The body shunts blood from non-vital organs to the vital organs.

The brain is a vital organ.

It receives increased blood volume.

In case of blood vessel aneurysm, the vessel, bursts; releasing blood into the brain tissue.

Hypertension increases afterload.

To compensate, the left ventricle increases the size of the wall.

The volume of the ventricle does not increase (Wenzel, Bode, Köhl, & Ehmke, 2016). Pathophysiology of Disorders sample assignment

Headache is a manifestation of hypertension. Patients suffer hemorrhagic strike as result of increased blood supply to brain through shunting and presence of  aneurysms.

Left ventricular hypertrophy is as a result of increased workload on the ventricle, thus the cardiac muscles stretch under the Sterling effects and hypertrophy to handle increased workload.

Factors

Genetic mutations in certain genes are associated

Age affects hypertension; which is common among elderly with hypertension

Age affects hypertension; which is common among elderly

Hypertension is more common among the female black population.

Lack of physical exercise behavior increases risk of hypertension.

Eating habits that comprises of fats causes obesity, risk factor for hypertension

Hypertension is diagnosed with a systolic blood pressure of above 140 mm Hg and a diastolic pressure of above 89 mm Hg (Sun, 2015). Pathophysiology of Disorders sample assignment.

Genetic make up determine the protein that make up the vascular walls. Therefore the elasticity and the vascular resistance. Vascular walls loose elasticity with aging. High fat content increase te deposition of fat on walls of the vascular lumen, increasing the resistance by affected blood vessels.

Treatment

The treatment of hypertension is done by use of several antihypertensive.

Calcium channel blocker reduce the effect of vasodilation.

Example is nifedipine

Beta blocker for instance; atenolol blocks the sympathetic stimulation.

Diuretics, for example furosemide increase renal water excretion.

Angiotensin converting enzyme , for example enalopril are another group of antihypertensive.

Central adrenergic receptor antagonists, for example methyldopa are antihypertensive.

Angiotensin II inhibitor, for example losartan is an antihypertensive (Blood Pressure Lowering Treatment Trialists’ Collaboration, 2014).

The treatment of hypertension relies on pharmacological effect of the drugs on the physiological mechanisms that control blood pressure. Pathophysiology of Disorders sample assignment.

Mind Map

Conclusion

In conclusion, hypertension is a common disorder.

The condition directly affects the cardiovascular. However, the pathophysiology affects multiple systems.

Hypertension is affected by several patient factors: age, genetics , gender , ethnicity and behavior.

The treatment of hypertension targets the pathways involved in pathophysiology.

The choice of treatment regime depends on severity of increased blood pressure.

References

Berkman, L. F., Kawachi, I., & Glymour, M. M. (Eds.). (2014).
Social epidemiology
. Oxford University Press.

Blood Pressure Lowering Treatment Trialists’ Collaboration. (2014). Blood pressure-lowering treatment based on cardiovascular risk: a meta-analysis of individual patient data.
The Lancet
,
384
(9943), 591-598.

Sun, Z. (2015). Aging, arterial stiffness, and hypertension.
Hypertension
,
65
(2), 252-256.

Wenzel, U. O., Bode, M., Köhl, J., & Ehmke, H. (2016). A pathogenic role of complement in arterial hypertension and hypertensive end organ damage.
American Journal of Physiology-Heart and Circulatory Physiology
,
312
(3), H349-H354.

Pathophysiology of Disorders sample assignment

Gastrointestinal Tract: Disorders of Motility Essay example

Assignment 1: Gastrointestinal Tract: Disorders of Motility

Jamie is a 3-month-old female who presents with her mother for evaluation of “throwing up.” Mom reports that Jamie has been throwing up pretty much all the time since she was born. Jamie does not seem to be sick. In fact, she drinks her formula vigorously and often acts hungry. Jamie has normal soft brown bowel movements every day and, overall, seems like a happy and contented baby. She smiles readily and does not cry often. Other than the fact that she often throws up after drinking a bottle, she seems to be a very healthy, happy infant. A more precise history suggests that Jamie does not exactly throw up—she does not heave or act unwell—but rather it just seems that almost every time she drinks a bottle she regurgitates a milky substance. Mom thought that she might be allergic to her formula and switched her to a hypoallergenic formula. It didn’t appear to help at all, and now Mom is very concerned.

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Cases like these are not uncommon. The mother was concerned and thinking her daughter may have an allergy; she changed to a different formula. However, sometimes babies have immature GI tracts that can lead to physiology reflux as they adapt to normal life outside the uterus. Parents often do not consider this possibility, prompting them to change formulas rather than seeking medical care. As in the case study above, GI alterations can often be difficult to identify because many cause similar symptoms Gastrointestinal Tract: Disorders of Motility Essay example. This same issue also arises with adults—adults may present with symptoms that have various potential causes. When evaluating patients, it is important for the advanced practice nurse to know the types of questions he or she needs to ask to obtain the appropriate information for diagnosis. For this reason, you must have an understanding of common GI disorders such as gastroesophageal reflux disease (GERD), peptic ulcer disease (PUD), and gastritis.

To Prepare

Review this week’s media presentation on the gastrointestinal system.

Review Chapter 35 in the Huether and McCance text. Identify the normal pathophysiology of gastric acid stimulation and production.

Review Chapter 37 in the Huether and McCance text. Consider the pathophysiology of gastroesophageal reflux disease (GERD), peptic ulcer disease (PUD), and gastritis. Think about how these disorders are similar and different.

Select a patient factor different from the one you selected in this week’s Discussion: genetics, gender, ethnicity, age, or behavior. Consider how the factor you selected might impact the pathophysiology of GERD, PUD, and gastritis. Reflect on how you would diagnose and prescribe treatment of these disorders for a patient based on this factor.

Review the “Mind Maps—Dementia, Endocarditis, and Gastro-oesophageal Reflux Disease (GERD)” media in the Week 2 Learning Resources. Use the examples in the media as a guide to construct a mind map for gastritis. Consider the epidemiology and clinical presentation of gastritis.

To Complete

Write a 2- to 3-page Gastrointestinal Tract: Disorders of Motility Essay example paper that addresses the following:

Describe the normal pathophysiology of gastric acid stimulation and production. Explain the changes that occur to gastric acid stimulation and production with GERD, PUD, and gastritis disorders.

Explain how the factor you selected might impact the pathophysiology of GERD, PUD, and gastritis. Describe how you would diagnose and prescribe treatment of these disorders for a patient based on the factor you selected.

Construct a mind map for gastritis. Include the epidemiology, pathophysiology, and clinical presentation, as well as the diagnosis and treatment you explained in your paper.

Notes

Please include a purpose statement

References must be 5 years or less

Please follow the rubric that will be uploaded.

Gastro-Intestinal Motility Disorders

Gastric acid is one of the common hormones that aid digestion and is produced in the stomach. Alteration in the stimulation and the production of gastric acid initiate a cascade of event that finally leads to disorders of gastro-intestinal motility.  Notably, some of the common disorders related to alteration in the production of gastric acid are: “gastro esophageal reflux disease (GERD), peptic ulcer disease (PUD), and gastritis” (Zatorski, 2017). The following  paper will present a discussion of the normal physiology of gastric acid production, development of the mentioned diseases based on gastric acid stimulation and production and impact of genetics on the development of the above three specified disorders. Finally, the paper will present a mind map of gastritis based on the epidemiological features, pathophysiological development, presentation, and management Gastrointestinal Tract: Disorders of Motility Essay example.

Gastric Acid Stimulation and Production

Gastric acid production is stimulated by endocrine and the nervous inputs. Gastric acid production is initiated in response to the presence of food in the stomach. When food enters the stomach, the G cells of pyloric mucosa produce gastrin hormone and is released into the stomach. Subsequently, through the process of churning in the stomach, the gastrin hormone comes into contact with the enterochromaffin-like cells of the gastric gland.  Upon activation by the hormone gastrin, enterochromaffin-like cells secrete histamine which, in turn, prompts the secretion of the acid by parietal cells. In addition to the hormonal stimulation, nervous stimulation leads to the production of gastric acid. The smell, taste or the sight of food causes the release of nerve impulses from the hypothalamus, the amygdala, and the cerebral cortex through the vagus nerve to the enterochromaffin-like cells in the stomach, causing the release of histamine which subsequently stimulates the secretion of gastric acid (Waldum, Kleveland, & Fossmark, 2015).

The histamine binds to H2 receptors on the enterochromaffin-like cells, activating the H+/ K+ ATPase proton pump. Chloride ions are actively transported from the cell’s cytoplasm into the canaliculi whereas sodium ions are pumped into the cell. As a result, the positively charged sodium and potassium ions bind to the negatively charged chloride ions, forming sodium chloride and potassium chloride in the cell’s canaliculi. In addition, within the cell’s cytoplasm, water is broken down into hydrogen and hydroxyl ions. The H+/ K+ ATPase proton pump transports the hydrogen ions from the cytoplasm of the parietal cells into the canaliculi of the cells by active transport. In turn, the negatively charged chloride ions bind with the positively charged chloride ions, forming gastric acid. Thereafter, the gastric acid flows into the lumen of the stomach, to aid in the activation of other digestive hormones (Schubert, 2017). Gastrointestinal Tract: Disorders of Motility Essay example.

Pathophysiology of GERD, Gastritis and PUD.

Notably, the level of gastric acid production is inhibited by several nervous and hormonal mechanisms. Therefore, the failure of the control mechanism initiates a cascade of events that lead to the development disorders such as GERD, PUD, and gastritis. Overproduction of gastric acid leads to the erosion of the mucosa of the lower end of that small intestine, a condition referred to as peptic ulcer disease. Additionally, in the event of the dysfunction of the pyrolic sphincter or a hiatal hernia, the reflux of gastric contents occurs. Particularly, in cases of overproduction of gastric acid, stomach contents with high levels of gastric acid flow to the esophagus, in a condition called gastro esophageal reflux disease. The high acidic contents cause irritation of the gastric and esophageal mucosa, causing gastritis and its complications (Schubert, 2017).

Genetics and Pathophysiology of GERD, PUD, and Gastritis

The genetic makeup of an individual determines the integrity of the pyloric sphincter and therefore plays a role in the development of gastro esophageal reflux disease. Subsequently, genetics play a role in the irritation of the mucosa of the stomach and esophagus. The presence of human leucocyte antigen (HLA) is genetically associated with the occurrence of peptic ulcer disease.  In general, genetics play a role in the occurrence and the prevalence of peptic ulcer disease, gastritis, and gastro-esophageal reflux disease Gastrointestinal Tract: Disorders of Motility Essay example. The association of genetics to the development of disorders of gastric motility, therefore, dictates on the treatment of the disorders. Particularly, during clinical management, it is imperative to take detailed family history concerning the occurrence of such conditions affecting family members so as to determine the causative factor and therefore the treatment. In addition, screening through genetic mapping to identify genetic markers associated with the development of any of the three disorders of gastric motility (Rugge, Fassan, & Graham, 2015).

In conclusion, the physiology of the stimulation and production of gastric acid is multifactorial and takes place through several stages. The alteration of the physiology of gastric acid initiates the pathophysiology of PUD, GERD and gastritis. Genetics play an important role in the development of gastritis and therefore the epidemiology, diagnosis, and treatment is significantly affected by the influence of genetics on gastritis. Gastrointestinal Tract: Disorders of Motility Essay example.

References

Rugge, M., Fassan, M., & Graham, D. Y. (2015). Epidemiology of gastric cancer. In
Gastric Cancer
Springer, Cham.

Schubert, M. L. (2017). Physiologic, pathophysiologic, and pharmacologic regulation of gastric acid secretion.
Current opinion in gastroenterology
,
33
(6), 430-438.

Waldum, H. L., Kleveland, P. M., & Fossmark, R. (2015). Upper gastrointestinal physiology and diseases.
Scandinavian journal of gastroenterology
,
50
(6), 649-656.

Zatorski, H. (2017). Pathophysiology and Risk Factors in Peptic Ulcer Disease. In
Introduction to Gastrointestinal Diseases Vol. 2
(pp. 7-20). Springer, Cham. Gastrointestinal Tract: Disorders of Motility Essay example.

Asthma sample essay assignment

Assignment: Asthma

Complications of asthma can be sudden. Consider the case of Bradley Wilson, a young boy who had several medical conditions. He appeared in good health when he went to school, returned home, and ate dinner. However, when he later went outside to play, he came back inside wheezing. An ambulance took him to the hospital where he was pronounced dead (Briscoe, 2012). In another case, 10-year-old Dynasty Reese, who had mild asthma, woke up in the middle of the night and ran to her grandfather’s bedroom to tell him she couldn’t breathe. By the time paramedics arrived, she had passed out and was pronounced dead at the hospital (Glissman, 2012). These situations continue to outline the importance of recognizing symptoms of asthma and providing immediate treatment, as well as distinguishing minor symptoms from serious, life-threatening symptoms Asthma sample essay assignment. Since these symptoms and attacks are often induced by a trigger, as an advanced practice nurse, you must be able to help patients identify their triggers and recommend appropriate treatment options. For this reason, you need to understand the pathophysiological mechanisms of chronic asthma and acute asthma exacerbation.

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To Prepare

Review “Asthma” in Chapter 27 of the Huether and McCance text. Identify the pathophysiological mechanisms of chronic asthma and acute asthma exacerbation. Consider how these disorders are similar and different.

Select a patient factor different from the one you selected in this week’s Discussion: genetics, gender, ethnicity, age, or behavior. Think about how the factor you selected might impact the pathophysiology of both disorders. Reflect on how you would diagnose and prescribe treatment of these disorders for a patient based on the factor you selected.

Review the “Mind maps—Dementia, Endocarditis, and Gastro-oesophageal Reflux Disease (GERD)” media in the Week 2 Learning Resources. Use the examples in the media as a guide to construct two mind maps—one for chronic asthma and one for acute asthma exacerbation. Consider the epidemiology and clinical presentation of both chronic asthma and acute asthma exacerbation Asthma sample essay assignment.

To Complete

Write a 2- to 3-page paper that addresses the following:

Describe the pathophysiological mechanisms of chronic asthma and acute asthma exacerbation. Be sure to explain the changes in the arterial blood gas patterns during an exacerbation.

Explain how the factor you selected might impact the pathophysiology of both disorders. Describe how you would diagnose and prescribe treatment for a patient based on the factor you selected.

Construct two mind maps—one for chronic asthma and one for acute asthma exacerbation. Include the epidemiology, pathophysiology, and clinical presentation, as well as the diagnosis and treatment you explained in your paper.

Note: The School of Nursing requires that all papers submitted include a title page, introduction, summary, and references. The Sample Paper provided at the Walden Writing Center provides an example of those required elements (available at http://writingcenter.waldenu.edu/57.htm). All papers submitted must use this formatting Asthma sample essay assignment.

Notes

Paper must include a purpose statement.

References must be 5 years or less

Please follow the rubric that will be uploaded.

Introduction

Asthma is a chronic inflammatory disease of the airway that is characterized by bronchial hypersensitivity and hyper-responsiveness to otherwise innocuous stimuli, resulting in reversible narrowing of the airway. Worldwide, it is estimated to occur in 334 million people (Asher, I, 2014). Asher’s study also lists Asthma as one of the top 10 causes of mortality in mid-childhood children.

Purpose

In this paper I will elucidate the pathophysiological mechanisms of chronic asthma and severe acute asthma and outline the management of these conditions with emphasis on symptomatic relief, preventing future attacks, and improving survival rates among asthmatics

Pathophysiology

Chronic asthma

The primary event that underlies the restructuring of the airway in chronic asthma is inflammation of the airway in response to commonly inhaled allergens such as those in dust, house mites, pollen or excrement from insects (Kudo, M, 2013.) This inflammation occurs in childhood and triggers type 2 T helper cells to secrete cytokines that mediate the inflammation. There is resultant mucosal edema, bronchial smooth muscle hypertrophy and mucous gland hypertrophy. These changes in the structure of the airway make it very responsive to subsequent stimuli with an exaggerated response.

The diagnosis of asthma is pegged on a high index of suspicion after taking a comprehensive history and physical examination. A history of cough, wheezing and chest tightness that are often triggered by cold, dust or exercise is very characteristic for asthma. These symptoms are usually reversible and the patient experiences period of normal respiration in between the attacks. Typically there is no fever accompanying the symptoms. The physical exam is often unremarkable unless there is an ongoing attack. A polyphonic wheeze may be heard on auscultation. The test of choice is assessment of lung function by spirometry Asthma sample essay assignment. A bronchial challenge test is used to diagnose asthma in a patient with normal lung function. The patient breathes increasing doses of nebulized methacholine with FEV
1
measurement. A normal subject is not expected to show significant dip in their FEV
1
unlike a patient with asthma. Lung inflation can also be demonstrated on a chest radiograph.

Acute asthma exacerbation

Severe acute asthma exacerbation, also referred to as status asthmaticus or near-fatal asthma, refers to the occurrence of bronchial obstruction that does not respond to the institution of standard bronchodilator therapy causing life threatening respiratory failure with marked hypoxemia and hypercapnia due to hampered ventilation (Nievas, I, 2013).

During the attack, there is marked mucosal edema. The remodeled airway has a relatively higher number of mucous producing cells and hypertrophied smooth muscles. The production of a lot of mucous and exaggerated active bronchoconstriction by the hypertrophied smooth muscles results in a very narrow airway that is plugged by mucous. A lot of energy is expended in an attempt to sustain adequate respiration. Air is trapped within the respiratory system with each inspiration. The patient will present with profound breathlessness, unable to complete sentences. On physical examination there will be tachypnea of more than 25 breaths per minute, tachycardia of more than 110 beats per minute and a pulmonary ejection fraction of less than 50%. The patient may have bradycardia, hypotension, cyanosis, confusion and may even go into a coma. Prompt reversal of this condition is necessary to prevent respiratory failure. Arterial blood gas analysis will demonstrate profound hypoxemia with hypocapnia because there is hyperventilation early on. After fatigue sets in, the PaC02 normalizes heralding impending respiratory failure (Nievas, I, 2013) Asthma sample essay assignment.

Treatment

Patient behavior is a critical predisposing factor to the development of acute asthma exacerbation. Asthmatic patients on follow up are usually put on both steroid and beta 2 agonist inhalers. The steroid component is prescribed to be taken on a daily basis to prevent airway remodeling by reducing the inflammatory response that goes on in the hypersensitive airway of the asthmatic patient. Lack of adherence to this management by patients allows the airway to remodel and ‘ready itself’ for an acute exacerbation (Taylor, A, 2014).

Emergency management of acute asthma exacerbation involves the administration of high flow oxygen if the saturation is below 92% and nebulization with a Beta 2 agonist. This is followed by the administration of intravenous then oral steroids. The patients are also often dehydrated therefore fluid resuscitation is necessary. There is a role for intravenous magnesium sulphate, ipratropium bromide, xanthines and antibiotics where there is concomitant infection. If the patient is still unresponsive to the treatment or develops a silent chest, ICU care will be necessary to provide ventilatory support Asthma sample essay assignment. In a patient with poor adherent behaviour, I will include a counselling session to their management to sensitize them on the need to be compliant with their medication so as to prevent future attacks.

Mind maps

Summary

The management of asthma remains to be a big challenge to nurse practitioners and physicians all over the world. Patient behavior compounds this problem when the patients show poor compliance to their preventive medication. It is imperative that patients become part of the management team by being adherent to their preventive medication hence reducing the incidence of acute asthma exacerbation. Asthma sample essay assignment.

References

Asher, I
. Pearce, N. (2014) Global burden of asthma among children
.
The       International Journal of Tuberculosis and Lung Disease
, Volume 18, Number 11, 1 November 2014, pp.
1269-1278(10)

Kudo, M., Ishigatsubo, Y., & Aoki, I. (2013). Pathology of asthma.
Frontiers in Microbiology
,
4
, 263.
http://doi.org/10.3389/fmicb.2013.00263

Nievas, I. F. F., & Anand, K. J. S. (2013). Severe Acute Asthma Exacerbation in Children: A Stepwise Approach for Escalating Therapy in a Pediatric Intensive Care Unit.
The Journal of Pediatric Pharmacology and Therapeutics : JPPT
,
18
(2), 88–104.
http://doi.org/10.5863/1551-6776-18.2.88

Taylor, A., Chen, L.-C., & Smith, M. D. (2014). Adherence to inhaled corticosteroids by asthmatic patients: measurement and modelling.
International Journal of Clinical Pharmacy
,
36
(1), 112–119. http://doi.org/10.1007/s11096-013-9862-0 Asthma sample essay assignment.

Disorders of the Veins and Arteries sample essay

Assignment: Disorders of the Veins and Arteries

Advanced practice nurses often treat patients with vein and artery disorders such as chronic venous insufficiency (CVI) and deep venous thrombosis (DVT). While the symptoms of both disorders are noticeable, these symptoms are sometimes mistaken for signs of other conditions, making the disorders difficult to diagnose. Nurses must examine all symptoms and rule out other potential disorders before diagnosing and prescribing treatment for patients. In this Assignment, you explore the epidemiology, pathophysiology, and clinical presentation of CVI and DVT.

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To Prepare

Review the section “Diseases of the Veins” (pp. 598-599) in Chapter 23 of the Huether and McCance text. Identify the pathophysiology of chronic venous insufficiency and deep venous thrombosis. Consider the similarities and differences between these disorders.

Select a patient factor different from the one you selected in this week’s Discussion: genetics, gender, ethnicity, age, or behavior. Think about how the factor you selected might impact the pathophysiology of CVI and DVT. Reflect on how you would diagnose and prescribe treatment of these disorders for a patient based on the factor you selected.

Review the “Mind Maps—Dementia, Endocarditis, and Gastro-oesophageal Reflux Disease (GERD)” media in the Week 2 Learning Resources. Use the examples in the media as a guide to construct two mind maps—one for chronic venous insufficiency and one for venous thrombosis. Consider the epidemiology and clinical presentation of both chronic venous insufficiency and deep venous thrombosis Disorders of the Veins and Arteries sample essay.

To Complete

Write a 2- to 3-page paper that addresses the following:

Compare the pathophysiology of chronic venous insufficiency and deep venous thrombosis. Describe how venous thrombosis is different from arterial thrombosis.

Explain how the patient factor you selected might impact the pathophysiology of CVI and DVT. Describe how you would diagnose and prescribe treatment of these disorders for a patient based on the factor you selected.

Construct two mind maps—one for chronic venous insufficiency and one for deep venous thrombosis. Include the epidemiology, pathophysiology, and clinical presentation, as well as the diagnosis and treatment you explained in your Disorders of the Veins and Arteries sample essay paper.

Note: The School of Nursing requires that all papers submitted include a title page, introduction, summary, and references. The Sample Paper provided at the Walden Writing Center provides an example of those required elements (available at http://writingcenter.waldenu.edu/57.htm). All papers submitted must use this formatting Disorders of the Veins and Arteries sample essay.

Introduction

The circulatory system constitutes of three main blood vessels including arteries, veins, and lymphatic. According to Bergan and Bunke, ( (2014), blood in the peripheral veins tends to be passive and under low pressure and since its pumping mechanism relies on muscle contraction in the legs and arms. However, physiological changes in the body lead to hypertension and trigger thrombi formation resulting in diseases of the veins. Notably, the clinical manifestations of common venous diseases may be misdiagnosed for other disorders. This paper provides a review of the pathophysiology of common vein diseases before concluding with mind maps highlighting the epidemiology, pathophysiology, clinical presentation, and treatment of Deep Vein Thrombosis(DVT)  and Chronic  Veins Insufficiency(CVI) Disorders of the Veins and Arteries sample essay.

Pathophysiology of Deep vein insufficiency

Deep vein insufficiency emanates from venous stasis. According to Eberhardt and Raffetto (2014),  the main factors resulting in venous stasis include a dysfunction in the venous valve, inflammation of the venous walls, dysfunctional pump mechanism, and venous hypertension. Notably, venous stasis results in an increase in blood viscosity and subsequent venous hypertension, factors which stimulate the release of vasoactive substances triggering the expression of adhesion molecules and inflammatory mediators. In this regard, the underlying cascade of events results in inflammation and subsequent rapture of peripheral veins walls, a condition imminent in CVI Disorders of the Veins and Arteries sample essay.

Pathophysiology of Deep Vein Thrombosis

Considering that the venous valves are vascular, the formation of microthrombi in the calf veins predisposes the development of DVT. Notably, venous stasis resulting from calf veins microthrombi obstruction and subsequent, endothelial hypoxemia stimulates the release of adhesion molecule and leukocytes (Huether and McCance, 2015). The leukocytes transfer the vasoactive molecules to the endothelial cells where they combine with activated Factor VII and through a cascade of events results to coagulation with the resultant formation of a thrombus. Di Nisio, Van and Büller (2016)) pinpoints that the main components of resultant venous thrombin are fibrin and erythrocytes. In this regard, clot formation results in the destruction of the peripheral veins, a condition imminent in Deep vein thrombosis Disorders of the Veins and Arteries sample essay.

Comparison of the pathophysiology of DVI and DVT

The pathophysiology of DVI and DVT harbor some similarities in the development of the underlying disorder. Notably, the development of both diseases involves three steps including venous stasis, coagulation activation, and subsequent endothelial damage in peripheral veins (Eberhardt and Raffetto, 2014). In this regard, there is a correlation between venous stasis, hemodynamic alterations, and hypertension in both disorders and their subsequent stimulation in the release of blood coagulation factors. Further, blood coagulation results in endometrial cells damage, a clinical manifestation evident in both DVI and DVT. Lastly, the mechanism of origin of venous stasis exhibit similarities with valve incompetence in peripheral veins of the body muscles is common in the progression of venous both venous diseases Disorders of the Veins and Arteries sample essay.

Differences between venous thrombosis and arterial thrombosis

The pathophysiology of venous and arterial thrombosis display notable differences. Di Nisio, Van and Büller (2016), asserts that despite thrombi formation, being a common manifestation in both disorders, their pathophysiology and outcome differs. Notably, arterial stasis results from plaque formation in the deep arteries and affects the valves in the left chambers of the heart. In contrast, venous stasis emanates from a variety of factors (both intrinsic and extrinsic) such as tissue damage and biochemical imbalance with its associated alterations affecting the soleus sinusoid and cusps valves in the extreme parts of the body Disorders of the Veins and Arteries sample essay. Additionally, the inflammatory response in arterial thrombosis results to coagulation involving the platelets while venous associated coagulation constitutes mainly fibrin and erythrocytes ( Di Nisio, Van  & Büller (2016). Finally, chronic inflammation in arterial thrombosis results in the acute coronary syndrome and ischemic stroke, while on the other hand, the pathogenesis of untreated venous thrombosis results to pulmonary embolism and deep vein thrombosis.

Pregnancy as a patient factor

Previous studies reveal that pregnancy is a risk factor in the development of DVI and DVT. According to (Khan, Vaillancourt, and Bourjeily (2017), the incidence of DVT among pregnant women is approximately 1.1 per 1000 deliveries. Further, risk increases during the last trimester and six weeks after parturition. In this regard, considering misdiagnosis of DVT and CVI may lead to the development asserts that there is a need for nurses and medical practitioners to undertake precautions in the diagnosis of the clinical manifestations of the diseases for effective treatment Disorders of the Veins and Arteries sample essay

Pathophysiology of DVT in pregnancy

Pregnancy results in anatomical and physiological changes in the body. According to Khan, Vaillancourt, and Bourjeily (2017), increased cardiac output resulting to peripheral vasodilation is a common event in pregnancy. Notably, the anatomical and physiological alterations associated with pregnancy result in venous stasis and hypercoagulability in the peripheral veins located in the legs and arms. In this regard, the body increases the production of procoagulant peptides and subsequently, decreases the release of the anticoagulant substances and as such, there is a formation of fibrin-rich thrombi, which initiates vascular damage of the endothelial cells in the peripheral veins located in legs and arms. Further, Khan, Vaillancourt, and Bourjeily (2017), asserts that vascular damage occurs in the pelvis during parturition

Diagnosis of DVT and CVI in pregnancy

Clinical diagnosis of DVT and DVI  should be accompanied with further laboratory tests to prevent misdiagnosis of the underlying disorder. Notably, a consideration of the physiological alterations associated with pregnancy should facilitate the decision-making process during the diagnosis of the disorders clinical manifestation. As such, the first criteria entail a physical examination of symptoms such as pain, tenderness, and swelling of the extremities in the patient with regard to the patient’s medical history (Khan, Vaillancourt, & Bourjeily 2017). However, since the clinical manifestations of DVT and pulmonary embolism in pregnancy are similar, I would recommend the patient to undergo an ultrasound scan analysis of the proximal venous system to confirm if the underlying condition is DVT Disorders of the Veins and Arteries sample essay.

Treatment

Considering that physiological changes in pregnancy result to hypercoagulation, administration of anticoagulants would be the first drugs of choice. In this regard, I would prescribe the patient to take low molecular weight heparin, which suppresses the release of clotting factors in the body. According to Khan, Vaillancourt, and Bourjeily (2017), LMWH has a safety profile since it does not cross the placental barrier and it reduces the risk of hemorrhage. Additionally, it does not affect the breast milk and it can be administered even after parturition.

Conclusion

Despite the fact that Diseases of the veins are common, their clinical manifestations tend to be misdiagnosed for other diseases. In this regard, there is a need for nurses and other medications to understand their pathophysiology for easy their treatment. Notably, understanding of the pathophysiology and clinical manifestations of these disorders will go a long way in facilitating the appropriate treatment for the underlying condition. Further, an understanding of the patient condition such as pregnancy will facilitate the adoption of a treatment option, which bests suits the patients Disorders of the Veins and Arteries sample essay.

Mind Map for Chronic Vein Insufficiency

M
ind Map For Deep Vein Thrombosis

References

Bergan, J. J., & Bunke, N. (Eds.). (2014). The vein book. Oxford University Press.

Di Nisio, M., van Es, N., & Büller, H. R. (2016). Deep vein thrombosis and pulmonary embolism. The Lancet, 388(10063), 3060-3073.

Eberhardt, R. T., & Raffetto, J. D. (2014). Chronic venous insufficiency. Circulation, 130(4), 333-346

Huether, S. E., & McCance, K. L. (2015). Understanding Pathophysiology-E-Book. Elsevier Health Sciences.

Khan, F., Vaillancourt, C., & Bourjeily, G. (2017). Diagnosis and management of deep vein thrombosis in pregnancy. BMJ: British Medical Journal (Online), 357 Disorders of the Veins and Arteries sample essay.

Review the section “Diseases of the Veins” (pp. 598-599) in Chapter 23 of the Huether and McCance text. Identify the pathophysiology of chronic venous insufficiency and deep venous thrombosis. Consider the similarities and differences between these disorders.

Select a patient factor different from the one you selected in this week’s Discussion: genetics, gender, ethnicity, age, or behavior. Think about how the factor you selected might impact the pathophysiology of CVI and DVT. Reflect on how you would diagnose and prescribe treatment of these disorders for a patient based on the factor you selected.

Review the “Mind Maps—Dementia, Endocarditis, and Gastro-oesophageal Reflux Disease (GERD)” media in the Week 2 Learning Resources. Use the examples in the media as a guide to construct two mind maps—one for chronic venous insufficiency and one for venous thrombosis. Consider the epidemiology and clinical presentation of both chronic venous insufficiency and deep venous thrombosis.

To Complete

Write a 3 page paper that addresses the following:

Compare the pathophysiology of chronic venous insufficiency and deep venous thrombosis. Describe how venous thrombosis is different from arterial thrombosis.

Explain how the patient factor you selected might impact the pathophysiology of CVI and DVT. Describe how you would diagnose and prescribe treatment of these disorders for a patient based on the factor you selected.

Construct two mind maps—one for chronic venous insufficiency and one for deep venous thrombosis. Include the epidemiology, pathophysiology, and clinical presentation, as well as the diagnosis and treatment you explained in your paper.

Please references should be within the last five years. All in text quotation should have paragraph number or page number. paper should have introduction and summary Disorders of the Veins and Arteries sample essay. Thank you

Disease of Arteries and veins

One of the conditions affecting the veins is deep venous thrombosis which affects 40% of women and 17 % of men globally and leads to pulmonary embolism, a life threatening condition. Valves are major anatomical and physiological components of the veins and chronic venous insufficiency results from impairment of the valves (Huether, & McCance, 2015). The discussion below will present a comparison of the pathophysiology of deep venous thrombosis, chronic venous insufficiency and arterial thrombosis. Finally, the essay will discuss how gender affects the pathophysiology, diagnosis and treatment of the above specified conditions and mind maps summarizing the conditions.

Pathophysiology

The predisposing factors to the development of the deep venous thrombosis include: female gender, pregnancy, overweight, cancer, immobility, post -partum period and surgical procedures. The predisposing factors in the development of chronic venous insufficiency include: long standing periods and deep venous thrombosis.  Chronic venous insufficiency basically refers to the impairment in the functioning of the valves that facilitate the uni-directional flow of blood through the veins to the heart. In contrast, deep venous thrombosis refers to a condition in which there is formation of a blood clot in the deep veins of the extremities. Chronic venous insufficiency affects both the deep and the superficial veins while the deep venous thrombosis affects the deep veins of the lower extremities in most of the cases. Deep venous thrombosis eventually leads to pulmonary thrombosis as a complication, whereas chronic venous insufficiency leads to ulceration and bacterial eczema following the effect the sequel of the condition on the walls of the affected veins. Deep venous thrombosis presents with tenderness and swelling of the affected lower limb whereas chronic venous insufficiency presents with hyperpigmentation, ulceration and varicose veins on the limb where the valves of the veins function insufficiently. Both the deep venous thrombosis and the chronic venous insufficiency are characterized by the occurrence of blood stasis in the affected veins. In addition, deep venous and chronic venous insufficiency present with pain and swelling of the affected extremities (Gloviczki, & Lawrence, 2017) Disorders of the Veins and Arteries sample essay.

Arterial thrombosis affects the arteries whereas deep venous thrombosis affects veins. In addition, arterial thrombosis affects any of the arteries whereas deep venous thrombosis affects the deep veins of the lower extremities in most cases and specifically is associated with the femoral vein, the popliteal vein or the ilio-femoral vein. The deep venous thrombosis is cause by injury to the endothelial lining of the veins which activates the coagulation pathway that involves conversion of prothrombin to thrombin and fibrinogen to fibrin. Finally a plug that consists of platelets, fibrin and red blood cells is formed (Gujja, Wiley, & Krishnan, 2014). In addition, blood stasis also causes the development of the deep venous thrombosis. Blood stasis is formed as result immobility.  Finally, a state hypercoagulability also leads to frequent and easy activation of the coagulation path way leading to a formation of the blood clot. On the other hand, arterial thrombosis is caused by the presence of thrombus, emboli, air emboli or cancer thrombi. Furthermore, deep venous thrombosis leads to the pulmonary thrombosis as a complication due to the mobility of the thrombi from the extremities, through the inferior vena cava to the heart and eventually to the lungs through the pulmonary artery. In contrast, arterial thrombosis leads to insufficient oxygen supply to the tissues that are supplied within blood by the affected artery. Subsequently, the body forms new collateral arteries to increase blood and oxygen supply to the affected tissues (Martinod, Witsch, Farley, Gallant, Remold‐O’Donnell, & Wagner, 2016).

Effect of Gender on DVT and DCI

Notably, the prevalence of the above mentioned conditions is higher among the women as compared to men.  In addition, pregnancy, post-partum period and the female gender are risk factors for the development of the deep venous thrombosis. During pregnancy the production of the clotting factors increases and thus, their concentration in the blood of the pregnant patient. Therefore, a state of hypercoagulability is created. Subsequently, following an injury to the endothelial lining of the blood vessel, the coagulation cascade is easily activated. In addition, reduced mobility is common among the pregnant women. Immobility contributes to the pathophysiological development of deep venous thrombosis and chronic venous insufficiency Disorders of the Veins and Arteries sample essay. Prolonged immobility reduces the contractility of the cuff muscles.  Therefore the propulsive force and effect of the muscles to the venous return through the major veins is affected. The diagnosis of deep venous thrombosis and chronic venous insufficiency should be guided by biographic data, medical and obstetric history of the patient. In cases of female patients, the clinician should be keen to take the obstetric history so as to elicit pregnancy and post -partum period as risks towards to the development of DVT and CVI. The treatment of DVT and CVI is universal regardless of the gender, and therefore anticoagulants and surgery are the treatment approaches (Aggarwal, Fullam, Brownstein, Maynard, Ansell, Varga, & Rickles, 2015).

                  
Mind Map

 
 
In conclusion, DVT and CVI are the most common conditions affecting the blood vessels. The two conditions differ widely in terms of the pathophysiology. However the conditions hare similarities. Gender affects the pathophysiology, prevalence and the diagnosis of DVT and CVI.

References

Aggarwal, A., Fullam, L., Brownstein, A. P., Maynard, G. A., Ansell, J., Varga, E. A., … & Rickles, F. R. (2015). Deep vein thrombosis (DVT) and pulmonary embolism (PE): awareness and prophylaxis practices reported by patients with cancer.
Cancer investigation
,
33
(9), 405-410.

Gloviczki, P., & Lawrence, P. F. (2017). Iliac vein stenting and contralateral deep vein thrombosis.
Journal of Vascular Surgery: Venous and Lymphatic Disorders
,
5
(1), 5-6.

Gujja, K., Wiley, J., & Krishnan, P. (2014). Chronic Venous Insufficiency.
Interventional cardiology clinics
,
3
(4), 5 Disorders of the Veins and Arteries sample essay93-605.

Huether, S. E., & McCance, K. L. (2015).
Understanding Pathophysiology-E-Book
. Elsevier Health Sciences.

Martinod, K., Witsch, T., Farley, K., Gallant, M., Remold‐O’Donnell, E., & Wagner, D. D. (2016). Neutrophil elastase‐deficient mice form neutrophil extracellular traps in an experimental model of deep vein thrombosis.
Journal of Thrombosis and Haemostasis
,
14
(3), 551-558 Disorders of the Veins and Arteries sample essay.

Cover Letter, Resume, and Portfolio Assignment

NURS 6660: Psychiatric Mental Health Nurse Practitioner Role I:

Child and Adolescent

Cover Letter, Resume, and Portfolio Assignment Guide

Your professional cover letter, resume, and portfolio are due by Day 7 of Week 10. It is highly

recommended that you begin planning and working on this Assignment as soon as it is viable.

The following checklists outline all of the items you should include in your cover letter, resume,

and portfolio. Additionally, the resources below have been provided to assist you in developing

your professional cover letter, resume, and portfolio.

Also, refer to the Walden University Career Center website for resources and develop your

cover letter and resume with the assistance of the Walden University Career Center website

for resources and make your cover letter, resume, and portfolio accessible online to your

faculty and potential employers.

BUY A PLAGIARISM-FREE PAPER HERE

Checklist for Cover Letter (S/U grade)

Your Cover Letter should be:

 Presented and formatted in professional business manner

 Addressed properly

 Clear and concise and include:

o Content Introduction

o Content Body

o Content Conclusion

 Written in a professional style and include:

o Correct spelling, punctuation, and grammar

o Clear and accurate sentence structure

Checklist for Resume (S/U) Cover Letter, Resume, and Portfolio Assignment

 Your Resume should be clear, concise and well-organized and it should also include

your:

 Name, address, business phone number, and email address (top center of resume)

 Profile: 2-3 sentences describing goal and positive attributes/characteristics

 Certifications & Licensure

 Education

 Professional Experience

 Honors/Awards

 References

© 2016 Laureate Education, Inc. 2 of 3

Checklist for Portfolio (S/U)

 Your Portfolio should be clear, concise and well-organized and it should also include

your:

 Personal Philosophy Statement

 Self-Assessment

 Personal goals (short term and long term)

 Achievements

 Cover Letter

 Resume

 Diplomas for formal education

 Letters of Recommendation

 References (list names, affiliation and contact information)

 Certifications

 Certificates of attendance for continuing education

 Transcripts

 Publications

 Oral Presentations and/or Poster Presentations

Cover Letter, Resume, and Portfolio Assignment Learning Resources

Resume, Portfolio, and Cover Letter Resources:

 Cover Letter Advice (n.d.). Nurse practitioner cover letter sample 1. Retrieved from

http://www.coverletter.us/nurse-practitioner-cover-letter/

 Dahring, R. (2013). Cover letter caveats. Retrieved from http://nurse-practitioners-andphysician-assistants.advanceweb.com/Columns/Career-Coach/Cover-Letter-Caveats.aspx

 NP Career Coach (n.d.). NP career coach resume tip sheet. Retrieved from

http://www.nursepractitionerjobsearch.com/nurse-practitioner-resume-tips.pdf

 Porche, D. J. & Danna, D. (2015). Cover letter & resume preparation: Every detail is

important when applying for a job. Advance healthcare network for NPs & PAs. Retrieved

from http://nurse-practitioners-and-physicianassistants.advanceweb.com/Features/Articles/Cover-Letter-Resume-Preparation.aspx

 Walden University Career Services (n.d.). Resumes & CVs: Home Retrieved from

http://academicguides.waldenu.edu/careerservices/resumesandcvs

© 2016 Laureate Education, Inc. 3 of 3

Portfolio Resources:

 Dennison, R. D. (2007). What goes into your professional portfolio and what you’ll get out of

it: Advance your nursing career with a professional portfolio. American Nurse Today 2(1).

Retrieved from

http://www.americannursetoday.com/assets/0/434/436/440/4352/4354/4368/4376/1b04d64f8cb0-4af6-adb6-7e3474d2603d.pdf

 Ferrara, S. (2013, October 31). Professional portfolio – A must have for NP students. [Blog

message]. Retrieved from http://onlinenursepractitionerprograms.com/2013/professionalportfolio-a-must-have-for-np-students/ Cover Letter, Resume, and Portfolio Assignment

 Hayes, E., Chandler, G., Merriam, D., & King, M. C. (2002). The master’s portfolio:

Validating a career in advanced practice nursing. Journal of the American Academy of

Nurse Practitioners, 14(3), 119.

 Walden University Career Services (2015). Quick start: Getting started with Optimal 2015.

Retrieved from https://youtu.be/Ir-B4MCuAvU

 Walden University Career Services (2015). Quick start: Getting started with Optimal

Resume: Presentation slides. Retrieved from

http://academicguides.waldenu.edu/ld.php?content_id=10634421

 Walden University Career Services (2015). Getting started with Optimal Resume: Tutorial.

Retrieved from http://academicguides.waldenu.edu/ld.php?content_id=10634430

 Walden University Career Services (2015). Getting started with Optimal Resume: Transcript.

Retrieved from http://academicguides.waldenu.edu/ld.php?content_id=10634440

 Weber, S. (2006). Developing nurse practitioner student portfolios. Journal of the American

Academy of Nurse Practitioners, 18(7), 301-302. doi:10.1111/j.1745-7599.2006.00134.x

FIND BELLOW MY CURRENT RESUME. FOR ANY FURTHER QUESTIONS PLEASE EMAIL ME AND I WILL RESPOND. THANKS IN ADVANCE

Victorine A Ngang

9937 whiskey run

Laurel, MD 20723

301-437-9937

05/11/2017

bebenav@yahoo.com

victorine.ngang@waldenu.edu

OBJECTIVE

Team-oriented registered nurse and a psychiatric mental health nurse practitioner student with a strong track record of professional driven ambition is seeking an internship opportunity for psychiatric mental health nurse practitioner role with adult and older adults. This clinical is geared towards medication prescription for adults and older adults Cover Letter, Resume, and Portfolio Assignment.

This clinical starts in February 2018 through May 2018 for a total of 144hours

EDUCATION AND TRAINING

¬¬-Walden University 2016-2018

Minneapolis, MN

Currently in school pursuing a Master in psychiatric nursing

Graduation date, 2018

-American Sentinel University 2015-2016

Denver, Colorado

Bachelors of Science in nursing

Graduation June, 2016

-Prince George’s Community College 2013

Largo, MD, USA

Associate of science in nursing

Graduation: August, 2013

Psychiatric Nurse Practitioner Internship

psychotherapy with individuals

Cumberland, MD

August 2017 – November 2017

Psychiatric Nurse Practitioner Internship

psychotherapy with groups and families

Cumberland, MD

November 2017 – February 2018

Psychiatric mental health NP role 1: Child

Columbia, MD

February 2018 – May 2018

EMPLOYMENT HISTORY

Registered Nurse

-Jessup Correctional institution (JCI): From April 2016 to present

• Provided nursing assessments and interventions including: medical, and psychiatric screening

• Observed and assessed changes and trends in physical or mental condition of the patient as well as planned and implemented therapeutic interventions and evaluate response to such interventions

• Administered medications, collect specimens, monitor and document treatment progress and patient response. Cover Letter, Resume, and Portfolio Assignment.

• Performed medical care for emergencies and prioritized care based on acuity

• Identified behavioral problems to make suitable recommendations.

• Provided therapeutic interactions with patients to assist them while in crisis.

• Administered medications and treatments as ordered by a physician.

-Registered nurse: From December 2013 to 2017

Ridgeway manor nursing and rehabilitation center- Catonsville, MD

• Perform health assessment, talking to patients about their problems and discussing the best way to plan and deliver their care.

• Caring for patients experiencing acute mental distress or who have an enduring mental illness.

• Provide health promotion, counseling and educating patients and families

• Administer medications, wound care, G-Tube care, IV care , tracheostomy care etc

• Interpret patient information and make critical decisions.

• Responsible for maintaining a safe environment through the use of monitoring devices.

• Provide social support for patients by creating and encouraging patients to take advantage of opportunities such as support groups, social activities and other therapeutic measures.

• Coordinate care, in collaboration with a wide array of healthcare professionals

• Direct and supervise care delivered by other healthcare personnel like LPNs,CNA and CMA.

LICENSURE / CERTIFICATION

-Current Registered Nurse Licensure in Maryland (RN)

-Cardio Pulmonary Resuscitation (CPR) Certified through American Heart Association Cover Letter, Resume, and Portfolio Assignment.